Dermakun (cats – 250 g)


For the symptomatic management of inflammatory skin conditions and poor hair quality.

  • Improves dermal barrier properties
  • Protects cell membranes from oxidative damage caused by inflammation
  • Minimizes inflammation and reduces itching
    • Inhibits the inflammatory response
    • Reduces histamine levels
  • Promotes wound-healing and the turnover of epithelial cells

Know more about

Available: 699 in stock


Flavoring byproduct (hydrolyzed pork liver and yeast), fish oil (sardine).

Nutritional additives

Natural products: Curcuma longa L.: Turmeric extract – 25 000 mg/kg; Flavourings: Piperine – 250 mg/kg; Vitamins: Vitamin C – 29 375 mg/kg, Calcium-D-pantothenate – 3 750 mg/kg, Vitamin E – 25 000 IU/kg, Biotin – 250 mg/kg, Niacinamide – 18 750 mg/kg; Trace elements: Zinc (zinc chelate of glycine) – 2 500 mg/kg.

Analytical components

Crude protein 20.35%; Crude fibres 0.00%; Crude fat 40.94%; Crude ash 9.01%; Omega 3 fatty acids 6.80%; Omega 6 fatty acids 1.40%

How do its active ingredients work?


Vitamin B3

It is essential in the formation of coenzyme NADPþ and takes part in biosynthetic reactions, such as in the synthesis of fatty acids, thus playing a key role in the synthesis of epidermal lipids.


Vitamin B5

It is present throughout the body in the form of coenzyme A. Coenzyme A plays a role in the formation of a number of essential fats and sterols. As such, it is important in the formation of the lipids that form the lamellar components of the skin barrier.



It acts as a cofactor of carboxylase synthetase enzymes. This group of enzymes catalyzes the biosynthesis of fatty acids, being this vitamin crucial in the maintenance of the dermal-barrier health.


Vitamin E

The inflammation generates a large amount of free radicals, which themselves accelerate the inflammatory process. Vitamin E is one of the most effective antioxidants for protecting polyunsaturated fatty acids of cell membranes from oxidation. Vitamin E inhibits lipid oxidation by scavenging peroxyl radicals much faster than these radicals can react with fatty acids or proteins from adjacent cell membranes.


Vitamin C

The excess of histamine aggravates inflammatory conditions of the dermis and respiratory tract. Most histamine is stored in tissue mast cells and blood basophils. After the release of histamine by these cells or by de novo synthesis, histamine triggers inflammation. Vitamin C spontaneously degrades histamine, being tissue histamine levels inversely related to vitamin C status.



Numerous reports have linked zinc deficiency to many dermatoses in dogs and cats, since it is needed for wound healing and the replacement of epithelial cells.Moreover, zinc deficiency accelerates the development of clinical signs due to lack of essential fatty acids, because it reduces ∆-6-desaturase enzyme activity. This enzyme is responsible for the synthesis of EPA and DHA from other simpler fatty acids.


Omega 3 fatty acids

They modulate eicosanoid production. Eicosanoids resulting from omega-3 fatty acid metabolism are less stimulants of the inflammatory response than those resulting from omega-6 fatty acids. Moreover, they are the precursors of resolvins and protectins, counter-regulatory inflammatory substances that serve as agonists for endogenous anti-inflammatory mechanisms and significantly act in the resolution of inflammation.



Its anti-inflammatory properties appears to be mediated through the inhibition of induction of important enzymes that mediate inflammatory processes like COX-2, LOX, and iNOS production of cytokines such as interferon-gamma and tumor necrosis factor, and activation of transcription factors like NF-κB and AP-1. Moreover, curcumin acts at multiple points, helping in enhancing wound-healing. Curcumin treatment results in faster reepithelialization of the epidermis, increased migration of myofibroblasts, fibroblasts, and macrophages in the wound bed, extensive neovascularization, and greater collagen deposition.



It increases curcumin bioavailability (by 20-fold), due to piperine’s inhibition of hepatic glucuronidation and intestinal metabolism of curcumin.

  • Atopic dermatitis that occurs with prurite and/or seborrea. In these cases, it allows reducing or event preventing the use of corticoids, anti-histaminics or immunomodulators.
  • Processes that occur with itching, redness, and inflammation of the skin.
  • Dermatoses due to nutrient deficiency (zinc, biotin, essential fatty acids or group B vitamins) that occur with coat of poor quality (dry, opaque, fragile, and brittle), generalized desquamation (seborrhea), crust formation in the friction areas, hair loss, lack of hair growth, and poor wound healing.
  • Situations of risk of zinc deficiency.
  • During shedding to improve hair quality and reduce excessive hair loss.


It is not recommended the use of DERMAKUN in case of:

  • Patients with clotting problems, co-administration with drugs that inhibit platelet-aggregation, or hemorrhagic processes.
  • Situations with subclinical iron deficiency or other diseases associated with iron.
  • Food allergy to fish or during treatment periods with exclusion diets.

Daily recommendation to be added to the food (on top). This dose can be divided in different shots:

Spoons* Amount for
0.5 2 months

*8.6 g per spoon


During how many time?

  • Severe atopic dermatitis: Administration is recommended for at least 15 days and as long as the symptoms persist.
  • Atopic dermatitis with controlled symptomatology: Reduce the daily dose up to the half as maintenance.
  • Seasonal atopic dermatitis: Begin the administration 15 days before the exposure to allergens.
  • Dermatosis due to nutrient deficiency: It is recommended to administer for 1 month for the resolution of symptoms. Then reduce the dose in half for maintenance and to avoid possible relapses.
  • Situations of risk of zinc deficiency: Administer intermittently during one month every other month as preventive treatment.
  • During shedding: Administration is recommended for at least 15 days and as long as shedding persist.